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Dihydrotestosterone (DHT) or 5α-dihydrotestosterone (5α-DHT), also known as 5α-androstane-17β-alcohol-3-one, is endogenous androgenic steroids and hormones. Enzyme 5α-reductase catalyzes the formation of DHT from testosterone in certain tissues, including the prostate, seminal vesicle, epididymis, skin, hair follicles, liver and brain. The enzyme mediates the reduction of testosterone C4-5 double bonds. DHT is quite effective as an agonist for androgen receptor (AR) relative to testosterone.
DHT can be used as a pharmaceutical preparation for use as a androgen or anabolic androgenic steroid (AAS). When used as a drug, it is called androgen ketone (INN) or sterane ketone (BAN). The availability of DHT is limited; it is not available in the United States or Canada, but can be used in certain European countries (including UK, Germany, France, Spain, Italy, Belgium and Luxembourg). DHT's brand names include Anaboleen, Anabolex, Anaprotin (UK), Andractel (formerly AndroGel-DHT) (FR, BE, LU), Androlone, Apeton, Gelovit (ES), Neodrol, Ophtovital, (DE), Pesomax (IT ), Stanaprol and Stanolone. Available formulations of DHT include oral or sublingual tablets (Anabolex, Stanolone), local gels (Andractim, Gelovit, Ophtovital), and esters as oils, injectable agents such as dihydrotestosterone propionate (pesomax) and valeric acid Dihydrotestosterone (Apeton). DHT esters are used as prodrugs for DHT in vivo and have long lasting drugs when administered intramuscularly. (Ermalone-Amp, Hermalone, Sarcosan) and dihydrotestosterone heptane (Anaboleen Depot) are additionally available DHT esters and others, including dihydrotestosterone acetate, dihydrotestosterone butyrate Ester and dihydrotestosterone formate, while developed but never listed.
Unlike testosterone and various synthetic AAS, DHT can not be fragrant and therefore does not present any dose of male breast development and other estrogen side effects risk. In addition, DHT can not be metabolized by 5α reductase (since it has been 5α-reduced) and therefore can not be enhanced in so-called androgen tissues such as skin, hair follicles and prostate. Compared with testosterone, this provides a greater proportion of exogenous DHT with anabolic and androgenic effects, and DHT may be less prone to certain skin and hair related side effects such as acne, oily skin, seborrhea, Hirsutism (excess facial / body hair growth) and androgenic hair loss (pattern hair loss), as well as prostate hyperplasia (which can lead to benign prostatic hyperplasia) and increase the risk of prostate cancer.
Drug DHT is mainly used for the treatment of male hypogonadism. A local preparation for the treatment of cachexia in cancer patients is being developed and the phase III clinical trial of the indications is reached, but is ultimately not introduced for this purpose. Although DHT itself has not been approved for the treatment of cachexia, the oral active synthetic derivative of DHT, oxandrolone (2-oxa-17a-methyl-DHT) is approved and used for this indication.
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